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FILE PHOTO: The Airbus A350 XWB is displayed during the ILA Berlin Air Show in Berlin, Germany, February 9, 2016. REUTERS/Axel Schmidt/File Photo

Airbus Chief Executive Tom Enders told Europe 1 radio on Tuesday that discussions with the industry about the needs of its customers would play a key role in determining the future of the world’s biggest aircraft program, which has been beset by setbacks and price increases.

“Discussions about the future of the project and the actual demand for the program will also come into play with the board,” Enders said. “We have this figure of around 280 units or even 330 units and we need the program to be successful.”

Airbus has revealed that the program faced a record $15 billion bill for delays and cost overruns over the last decade, and that it now expected losses of between 2 billion and 4.5 billion euros ($2.3-4.6 billion) this year.

Airbus aims to start flying the new plane, the largest that it has ever built, in late 2018 after six delays for a delivery date of 2017.

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The wings of the Airbus A350 XWB, which can be made

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Leukocytes and antibody-secreting B lymphocytes play a role in the earliest stage of atherogenesis. In a rabbit model of vascular injury, leukocytes remained at the site of injury while B lymphocytes moved from the perivascular space to the subendothelial space where they transformed into plasma cells which secrete IgG against specific vascular antigens. Immunization of rabbits with oxidized low-density lipoproteins (LDL) to which they had been sensitized induced both leukocytic and lymphocytic inflammation. In multiple regression analyses, high plasma IgG levels, high LDL levels, and hyperlipidemia were independent predictors of the severity of atherosclerosis. In humans, IgG was present in the intima prior to any evidence of atherogenic lesion development. IgG-secreting cells were prominent in the earliest stage of lesion development and commonly located in a subendothelial position. IgM, probably the isotype of IgG, was present in the intima of only a few patients. However, in mice, IgM coated the deposits in advanced atherosclerotic lesions. The complex mode of action of IgG in the early stages of atherogenesis seems to be related to the ability of the lipoprotein to induce cytokines and induce chemokines which attract leukocytes. The ability of IgG to bind to and activate complement may also be related to local cytokine levels.Rationale of taking an implant-free interval before initiating definitive fixation in diaphyseal radial fracture.
A detailed understanding of the factors contributing to implant failure is fundamental to the decision making process. The aim of this study was to identify the main factors associated with non-unions in patients with diaphyseal radial fractures treated with open reduction and internal fixation (ORIF). A prospective cohort of 100 consecutive patients with diaphyseal fractures of the radius treated with ORIF with delayed internal fixation was formed. Clinical follow up at a minimum of 12 months was performed. The main factors that could predispose to a poor outcome were identified from the recorded data. The primary endpoint


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